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2015). These studies, taken together, counsel that, Despite the fact that a direct activation of GPR55 or PPAR‐α happens, PEA can deliver its anti‐inflammatory motion during the gut also by using indirect activation of CB1 and CB2 receptors, likely as a consequence of the ability of this compound to potentiate the motion of endocannabinoids at
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2005). These results weren't shocking if we look at that PEA is a badly drinking water‐soluble material, that may limit its oral absorption and bioavailability, Nonetheless they may suggest a brief‐lived motion of PEA, in settlement with The reality that this compound is degraded by two distinctive hydrolases, that may be, NAAA and FAAH. Later